Publisher's Synopsis
In previous work the author proposed an endocrine disruption theory of schizophrenia in which an abnormal exposure to prenatal estrogen programs the fetus to develop schizophrenia later in life. This book expands the theory by discussing how several risk factors of schizophrenia can be explained by endocrine disruption. The book discusses endocrine disruption in general, how it challenges certain traditional concepts of toxicology, and may cause or contribute to several human diseases. The most recent information regarding the potential role of the estrogenic endocrine disruptor bisphenol-A in neurodevelopment is examined. Risk factors for schizophrenia including genetics, obstetric complications, famine, lead poisoning, and viral infections are discussed within the context of estrogenic endocrine disruption as the pathological root cause.
